Exercise protects against reperfusion arrhythmia by sustaining mitochondrial energetics (881.1)
نویسندگان
چکیده
منابع مشابه
Exercise-induced protection against reperfusion arrhythmia involves stabilization of mitochondrial energetics.
Mitochondria influence cardiac electrophysiology through energy- and redox-sensitive ion channels in the sarcolemma, with the collapse of energetics believed to be centrally involved in arrhythmogenesis. This study was conducted to determine if preservation of mitochondrial membrane potential (ΔΨm) contributes to the antiarrhythmic effect of exercise. We utilized perfused hearts, isolated myocy...
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ژورنال
عنوان ژورنال: The FASEB Journal
سال: 2014
ISSN: 0892-6638,1530-6860
DOI: 10.1096/fasebj.28.1_supplement.881.1